%100 İnce mikrofiber den üretilmiştir.

NASA mühendisleri tarafından geliştirilen tek patentli anti-mite kumaştır.

(The Microair Pristine® mattress encasing is made with the tight weave fabric of the same name which has revolutionised the world of anti-mite covers. Pristine, composed of a very high number of polyester microfibers in a warp and weft structure, creates a total barrier against mites and their allergens. It is the only patented anti-mite fabric (Patent No. US 6,277,770 B1) because it was designed by NASA engineers to perform this function with maximum comfort.)

Klinik çalışmalar ile kanıtlanmış tek üründür.

- a comparative study of 32 mite allergen-impermeable encasings from 9 countries to find which ones are really effective (Mahakittikun et al, Mite penetration of different types of material claimed as mite proof by the Siriraj chamber method, J Allergy Clin Immunol 2006;118:1164-8);

- a comparative study of mite allergen-impermeable encasings in non-woven fabric and woven dust mite proof encasings(Pristine
® patent) - (Miller J.D. et al, Nonwoven in contrast to woven mattress encasings accumulate mite and cat allergen, J Allergy Clin Immunol 2007, volume 120, number 4, pages 977-979);

- a laboratory test demonstrating that Pristine blocks more than 99% of the dust mite allergens commonly present in mattresses and pillows (IBT Laboratories - Specializing in molecular & cellular immunology).

-November 2006Volume 118, Issue 5, Pages 1164-1168

Mite penetration of different types of material claimed as mite proof by the Siriraj chamber method


Clin Exp Allergy. 1998 Dec;28(12):1487-92.

House dust mite allergen exposure in infancy.

Mahmic A1Tovey ERMolloy CAYoung L.

Author information



Infancy may be a critical time for exposure to house dust mite allergens, when exposure to high levels can increase the risk of allergic sensitization and the development of asthma in later life.


To measure house dust mite allergen (Der p 1) concentration in the infants' environment and examine lifestyle factors which may influence mite allergen exposure.


Infants aged between 4 and 12 months (n = 134) from the western region of Sydney, Australia. participated. Reservoir dust samples were collected from four sites within each home: infant's bed, second bed (adult or second child's bed), lounge floor and sheepskins (where available). Settling aeroallergen was collected for 10-14 d in Petri dishes in the infant's room. Der p 1 was measured by ELISA. A questionnaire on types of bedding, sleeping and playing patterns of the infant was completed by the parents at the time of dust collection.


All infants were exposed to at least one site with Der p 1 concentrations greater than 10 microg/g fine dust. The mean settling aeroallergen level in the infants' room was 24 ng De p l/m2 day and this was weakly related to bed allergen levels (r=0.21, P<0.05). Mattress type had a weak effect on Der p 1 levels as measured in the whole bed (P = 0.07), while bed cover and bed type had no effect (P>0.6). The mean product of time spent at a site and its allergen concentration was highest for beds in 69% of infants.


The high level of allergen exposure in the environment of this group of infants places them at an increased risk of early sensitization and development of asthma. Any strategy to reduce asthma prevalence should address these high and avoidable levels.


- Pediatr Allergy Immunol. 2005 Feb;16(1):27-31.

Prenatal exposure to mite and pet allergens and total serum IgE at birth in high-risk children.

Schönberger HJ1Dompeling EKnottnerus JAKuiper Svan Weel CSchayck CP.

Author information


To examine the relationship between prenatal exposure to mite, cat and dog allergens and total serum IgE at birth in newborns at high risk of asthma. In the homes of 221 newborns with at least one first-degree relative with asthma, concentrations (ng/g dust) of allergens of house dust mite (mite), cat and dog were measured at the fourth to sixth month of pregnancy in dust samples from the maternal mattress and living room. At day 3-5 after birth, total IgE was measured in capillary heel blood. A total number of 174 blood samples were available (11 mothers refused newborn's blood sampling, and in 36 cases the blood sample was too small for analysis). In 24% of the newborns, total IgE was elevated (cut-off value 0.5 IU/ml). A significant dose response relationship was found between increasing mite allergen levels [divided in quartiles ng/g dust (qrt)] and the percentage of elevated IgE: first qrt (0-85 ng/g) 13%; second qrt (86-381) 19%; third qrt (382-2371) 26%; fourth qrt (> or =2372) 42%, respectively, p=0.01. This relationship remained significant after adjusting for passive smoking, maternal and paternal mite allergy, socio-demographic factors, birth characteristics and (breast) feeding practice in the first week of life. In high-risk newborns, prenatal exposure to mite allergens, but not to cat and dog allergens from dust of the living room and of the maternal mattress was associated with total serum IgE at birth.