İnce mikrofiber den üretilmiştir.
mühendisleri tarafından geliştirilen tek patentli anti-mite kumaştır.
Microair Pristine® mattress
encasing is made with the tight weave fabric of the same name which has
revolutionised the world of anti-mite covers. Pristine,
composed of a very high number of polyester microfibers in a warp and weft
structure, creates a total barrier against mites and their allergens. It is the
only patented anti-mite fabric (Patent No. US 6,277,770 B1) because it was
designed by NASA engineers to perform this function with maximum comfort.)
çalışmalar ile kanıtlanmış tek üründür.
- a comparative study of 32 mite
allergen-impermeable encasings from 9 countries to find which ones are really
effective (Mahakittikun et al, Mite penetration of
different types of material claimed as mite proof by the Siriraj chamber
method, J Allergy Clin Immunol 2006;118:1164-8);
comparative study of mite allergen-impermeable encasings in non-woven fabric
and woven dust mite proof encasings(Pristine® patent) -
(Miller J.D. et al, Nonwoven in contrast to woven mattress encasings accumulate
mite and cat allergen, J Allergy Clin Immunol 2007, volume 120, number 4, pages
laboratory test demonstrating that Pristine blocks more than 99% of the dust
mite allergens commonly present in mattresses and pillows (IBT Laboratories - Specializing in molecular
& cellular immunology).
118, Issue 5, Pages 1164-1168
Mite penetration of different types of material
claimed as mite proof by the Siriraj chamber method
Clin Exp Allergy. 1998 Dec;28(12):1487-92.
dust mite allergen exposure in infancy.
Mahmic A1, Tovey ER, Molloy CA, Young L.
may be a critical time for exposure to house dust mite allergens, when exposure
to high levels can increase the risk of allergic sensitization and the
development of asthma in later life.
measure house dust mite allergen (Der p 1) concentration in the infants'
environment and examine lifestyle factors which may influence mite allergen exposure.
aged between 4 and 12 months (n = 134) from the western region of Sydney,
Australia. participated. Reservoir dust samples were collected from four sites
within each home: infant's bed, second bed (adult or second child's bed), lounge
floor and sheepskins (where available). Settling aeroallergen was collected for
10-14 d in Petri dishes in the infant's room. Der p 1 was measured by ELISA. A
questionnaire on types of bedding, sleeping and playing patterns of the infant
was completed by the parents at the time of dust collection.
infants were exposed to at least one site with Der p 1 concentrations greater
than 10 microg/g fine dust. The mean settling aeroallergen level in the
infants' room was 24 ng De p l/m2 day and this was weakly related to bed
allergen levels (r=0.21, P<0.05). Mattress type had a weak effect on Der p 1
levels as measured in the whole bed (P = 0.07), while bed cover and bed type
had no effect (P>0.6). The mean product of time spent at a site and its allergen
concentration was highest for beds in 69% of infants.
high level of allergen exposure in the environment of this group of infants
places them at an increased risk of early sensitization and development of
asthma. Any strategy to reduce asthma prevalence should address these high and
- Pediatr Allergy
exposure to mite and pet allergens and total serum IgE at birth in high-risk
Schönberger HJ1, Dompeling E, Knottnerus JA, Kuiper S, van Weel C, Schayck CP.
To examine the relationship between prenatal
exposure to mite, cat and dog allergens and total serum IgE at birth in
newborns at high risk of asthma. In the homes of 221 newborns with at least one
first-degree relative with asthma, concentrations (ng/g dust) of allergens of
house dust mite (mite), cat and dog were measured at the fourth to sixth month
of pregnancy in dust samples from the maternal mattress and living room. At day
3-5 after birth, total IgE was measured in capillary heel blood. A total number
of 174 blood samples were available (11 mothers refused newborn's blood
sampling, and in 36 cases the blood sample was too small for analysis). In 24%
of the newborns, total IgE was elevated (cut-off value 0.5 IU/ml). A
significant dose response relationship was found between increasing mite
allergen levels [divided in quartiles ng/g dust (qrt)] and the percentage of
elevated IgE: first qrt (0-85 ng/g) 13%; second qrt (86-381) 19%; third qrt
(382-2371) 26%; fourth qrt (> or =2372) 42%, respectively, p=0.01. This
relationship remained significant after adjusting for passive smoking, maternal
and paternal mite allergy, socio-demographic factors, birth characteristics and
(breast) feeding practice in the first week of life. In high-risk newborns,
prenatal exposure to mite allergens, but not to cat and dog allergens from dust
of the living room and of the maternal mattress was associated with total serum
IgE at birth.